| 货号 | 13571-50ug |
| 描述 | The metabolism of F series prostaglandins (PGs), including PGF1α and PGF2α, commonly begins with the reduction of the double bond between C-13 and C-14 and oxidation of the hydroxyl group at C-15, producing 13,14-dihydro-15-keto PGs. The removal of four carbons at the α-terminus and oxidation of the terminal ω carbon produces the abundant urinary metabolites, including tetranor-PGFM.1,2,3,4 13,14-dihydro-15-keto tetranor PGF1α is a potential metabolite of either PGF1α or PGF2α and likely precursor to tetranor-PGFM. |
| 供应商 | Cayman |
| 应用文献 | |
| 1.Granström, E., and Samuelsson, B. The structure of a urinary metabolite of prostaglandin F2α in man. Journal of the American Chemical Society 91, 3398-3400 (1969). 2.Granström, E., and Samuelsson, B. On the metabolism of prostaglandin F2α in female subjects. The Journal of Biological Chemisty 246, 5254-5263 (1971). 3.Hamberg, M. Quantitative studies on prostaglandin synthesis in man III. Excretion of the major urinary metabolite of prostaglandins F1α and F2α during pregnancy. Life Sciences 14, 247-252 (1974). 4.Hamberg, M. Quantitative studies on prostaglandin synthesis in man II. Determination of the major urinary metabolite of prostaglandins F1α and F2α. Analytical Biochemistry 55, 368-378 (1973). | |
| 运输条件 | Room temperature in continental US; may vary elsewhere |
| 存放说明 | -20 |
| 纯度 | ≥98% |
| 计算分子量 | 300.4 |
| 分子式 | C16H28O5 |
| CAS号 | 24379-94-0 |
| 稳定性 | ≥ 1 year |
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